Saturday, November 30, 2013

Answer 20

20.  Choice A is the correct answer.  This patient has the typical ST depression in the septal leads V1 and V2.  This patient is also has ST elevation in the inferior leads (II, III, and aVf) suggestive of an inferior wall MI.  There is no ST depression in the lateral leads (I, avL, V5 and V6)




Question 20

20.  Your patient is a 72 year old male with chest pain and shortness of breath.  His EKG is below.  Which of the following is the correct diagnosis?


A.  Posterior Wall and Inferior Wall MI
B.  Septal Ischemia
C.  Inferior Wall MI
D.  Lateral Wall ischemia

Answer 19

19.  Choice D is the correct answer.  Dihydropyridines dilate arterioles only (Nifedipine, Amlodipine, and Felodipine) and Non Dihydropyridines slow calcium channels in the heart and dilates arterioles (verapamil and diltazem).




Question 19

19.  What is the difference between dihydropyridines and nondihydropyridines?

A.  Dihydropyridines slow down calcium channels in the heart and nondihydropyridines do not

B.  Dihydropyridines constrict arterioles

C.  Both dihydropyridines dilate arterioles and nondihydropyridines constrict arterioles

D.  Nondihydropyridines slow calcium channels in the heart and dilate arterioles


Answer 18

18.  Choice B is the correct answer.  Patients with ACE inhibitor induced angioedema are typically deficient in C1 Esterase.  Treatment involves Fresh Frozen Plasma.  Corticosteroids and antihistamines should be administered also.  Patients with airway issues should be intubated until this can be treated.  Prostaglandins and bradykinnin are increased in patients taking ACE inhibitors.  TNF has nothing to do with this disease process.




Question 18

18.  Your patient is a 45 year old male that presents with the swelling below.  He has been taking lisinopril for hypertension.  Which of the following enzymes deficiency is likely responsible for the swelling?



A.  Prostaglandins
B.  C1 Esterase
C.  Bradykinnin
D.  Tumor Necrosis Factor (TNF)

Answer 17

17.  Choice D is the correct answer.  Cyanide poisoning is not a cause of a cause of secondary hypertension.  Lead poisoning is.  Pheochromocytoma, porphyria, and hyperthyroidism/hypothyroidism are all causes of secondary hypertension.